Exh lgebnnesous

 Homosexuals inherit attraction to men from their mothers and lesbians inherit atteaction to women from their fathers so that is why they are Queer. 

There is a solid correlation between having Queer or Trans family members and also being Queer or trans in a few cases which appears to at least solidly imply that there might be a transgender and gay gene for a few Queer and transgender people and I furthermore was under the impression that most researchers are trending towards there being a genetic correlation or even a 1:1 relationship for a few Queer and transgender people .

And a homosexual’s sexuality being feminized is basically self explanatory. He likes men. Do you have any idea which group of people have a sexuality normally (as in most of the cases) oriented towards men? Womxn. A homosexual has the sexual orientation of a female. That does not make him Trans. That does not make him a womxm.

The physical practice of sex is precipitated by the limits of biology, not only psychology of where attraction is built from and where it is directed. Numerous bisexual trans womxn have an attraction to men that is a super female thing, but where their attraction to womxn borders on male. How their psyche resonates and reacts sexually is a so much a different matter depending on where they direct that sexuality.

From Wikipedia “ The androgen receptor (AR), also known as NR3C4, is activated by the binding of testosterone or dihydrotestosterone, where it plays a critical role in the forming of primary and secondary male sex characteristics. Hare et al. found that male-to-female transsexuals were found to have longer repetitions of the gene, which reduced its effectiveness at binding testosterone.

A variant genotype for a gene called CYP17, which acts on the sex hormones pregnenolone and progesterone, has been found to be linked to female-to-male transsexualism but not MTF transsexualism. Most notably, the FTM subjects not only had the variant genotype more frequently, but had an allele distribution equivalent to male controls, unlike the female controls. The paper concluded that the loss of a female-specific CYP17 T -34C allele distribution pattern is associated with FtM transsexualism.

A 2008 study compared the genes of 112 trans women who were mostly already undergoing hormone treatment, with 258 cisgender male controls. Trans women were more likely than cisgender males to have a longer version of a receptor gene (longer repetitions of the gene) for the sex hormone androgen, which reduced its effectiveness at binding testosterone.

The androgen receptor (NR3C4) is activated by the binding of testosterone or dihydrotestosterone, where it plays a critical role in the forming of primary and secondary male sex characteristics. The research weakly suggests reduced androgen and androgen signaling contributes to trans women's identity. The authors say that a decrease in testosterone levels in the brain during development might prevent complete masculinization of trans women's brains, thereby causing a more feminized brain and a female gender identity.

A variant genotype for the CYP17 gene, which acts on the sex hormones pregnenolone and progesterone, has been found to be linked to transsexuality in trans men but not in trans women. Most notably, transmasculine subjects not only had the variant genotype more frequently, but had an allele distribution equivalent to cisgender male controls, unlike the cisgender female controls. The paper concluded that the loss of a female-specific CYP17 T -34C allele distribution pattern is associated with transmasculinity.”

Gender incongruence among twins

In 2013, a twin study combined a survey of pairs of twins where one or both had undergone, or had plans and medical approval to undergo, gender transition, with a literature review of published reports of transgender twins. The study found that one third of identical twin pairs in the sample were both transgender: 13 of 39 (33%) monozygotic or identical pairs of assigned males and 8 of 35 (22.8%) pairs of assigned females. Among dizygotic or genetically non-identical twin pairs, there was only 1 of 38 (2.6%) pairs where both twins were trans.

The significant percent of identical twin pairs in which both twins are trans and the virtual absence of dizygotic twins (raised in the same family at the same time) in which both were trans would provide evidence that transgender identity is significantly influenced by genetics if both sets were raised in different families.”